CJC-1295 with DAC

CJC-1295 DAC is a synthetic growth hormone-releasing hormone (GHRH) analogue consisting of the first 29 amino acids of GHRH, conjugated with a drug affinity complex (DAC) that extends its plasma half-life to approximately 6-8 days.

CJC-1295 is a synthetic analogue of growth hormone-releasing hormone (GHRH) that increases plasma levels of growth hormone (GH) and insulin-like growth factor 1 (IGF-1). The addition of a drug affinity complex (DAC) via a lysine linker allows the peptide to bind to albumin in the bloodstream, extending its half-life from minutes to approximately 6-8 days.

Overview

Growth hormone-releasing hormone (GHRH) has been modified to produce analogues that preserve certain physiologic effects while improving pharmacokinetic properties. CJC-1295 consists of the first 29 amino acids of GHRH (a maleimido derivative of hGRF(1-29)) and was developed to create a more soluble GH secretagogue that is easier to manufacture in quantity while preserving the physiologic actions of native GHRH.

CJC-1295 was originally developed by ConjuChem Biotechnologies with the DAC moiety included, and the combination was simply called "CJC-1295." When the peptide began to be offered without DAC, naming confusion arose. The convention now is: CJC-1295 DAC refers to the molecule with the drug affinity complex, while CJC-1295 (or CJC-1295 no-DAC) refers to the peptide without it.

Mechanism of Action

CJC-1295 acts on the GHRH receptor (GHRH-R) on anterior pituitary somatotroph cells, stimulating the synthesis and pulsatile release of growth hormone. The DAC component binds to albumin via a reactive chemical group, forming a stable bioconjugate that circulates for days rather than minutes.

Key mechanistic features:

  • Preserved pulsatility -- Unlike exogenous GH administration, CJC-1295 DAC maintains the normal circadian peaks and valleys of endogenous GH secretion (Ionescu & Frohman, 2006).
  • Extended half-life -- The DAC-albumin conjugation extends half-life from minutes (native GRF) to approximately 6-8 days.
  • IGF-1 elevation -- Downstream IGF-1 levels increase proportionally to GH output, mediating many of the anabolic effects.

Reconstitution Calculator

CJC-1295 with DAC

CJC-1295 is a synthetic analogue of growth hormone-releasing hormone (GHRH) that

Draw Volume
1.000mL
Syringe Units
100units
Concentration
1,000mcg/mL
Doses / Vial
2doses
Vial Total
2mg
Waste / Vial
0mcg
Syringe Cap.
100units · 1mL
Recommended Schedule
M
T
W
T
F
S
S
FrequencyOnce or twice weekly
TimingSubcutaneous injection, spaced 3-4 days apart if twice week…
Cycle8-12 weeks
NoteClinical study (Teichman et al., JCEM 2006, PMID: 16352683) used 30-60 mcg/kg SC weekly/biweekly.
How to reconstitute
Gather & prepare
1/6Gather & prepare

Set up a clean workspace with all supplies ready.

1.Wash hands thoroughly, put on disposable gloves
2.Your 2mg peptide vial (lyophilized powder)
3.Bacteriostatic water (you'll need 2mL)
4.A 3–5mL syringe with 21–25 gauge needle for reconstitution
5.Alcohol swabs (70% isopropyl)
Use bacteriostatic water (0.9% benzyl alcohol) for multi-dose vials. Sterile water is only safe for single-use.
Supply Planner

2x / week for weeks

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4vials
8 doses7 days/vial
Cost Breakdown
Vial price
$0.00per dose
$0.00 /week$0 /month
Store 2-8°C30 day shelf lifeSwirl gentlyFor research purposes only

Research

Growth Hormone Release

A single dose of CJC-1295 increases GH levels by 2-10 fold in animal models. Peak GH levels are reached approximately 2 hours after administration and effects persist for about six days. Critically, CJC-1295 preserves the normal pulsatile pattern of GH release, meaning circadian peaks and valleys remain intact. This physiologic secretion pattern avoids the side effects associated with continuous GH elevation and preserves downstream effects on hyperplasia, hypertrophy, bone density, blood sugar, fat metabolism, protein synthesis, and myocardial function (Ionescu & Frohman, 2006).

Growth Normalization

Studies in GHRH knockout mice demonstrated that once-daily administration of CJC-1295 normalizes GH release and growth patterns. This finding makes CJC-1295 potentially useful as a therapy for growth abnormalities in various mammalian species. Preserving normal pulsatile GH release is critical for appropriate downstream signaling, particularly through IGF-1 (Alba et al., 2006).

Fertility

Research from the early 1990s suggests that CJC-1295 and other GRF analogues may promote ovulation in infertile patients. Ovulation is dependent on IGF-1 and is influenced by GH release cycling. Animal models of superovulation have confirmed that both GH and IGF-1 levels rise significantly around the time of ovulation, and exogenous administration of GH secretagogues is sufficient to induce appropriate ovulation (Volpe et al., 1991). There is speculation that CJC-1295 may also be useful in male infertility through regulation of spermatogenesis, though this remains untested.

Ongoing & Future Research

CJC-1295 DAC is not currently in active clinical development following the discontinuation of ConjuChem's program. However, the broader GHRH analog class remains an active area of research:

  • NCT00071240 -- Phase 2 study of CJC-1295 (ConjuChem) evaluating pharmacokinetics and GH/IGF-1 response in healthy adults. Completed; provided the foundational PK data.
  • NCT00935493 -- Tesamorelin (GHRH analog) Phase 3 study in HIV lipodystrophy, demonstrating the clinical viability of the GHRH analog class.
  • NCT02696642 -- Study of tesamorelin effects on liver fat in HIV, extending GHRH analog applications to hepatic steatosis.
  • NCT03375112 -- Tesamorelin for peripheral neuropathy in prediabetes and diabetes, exploring neurotrophic effects of GHRH-pathway stimulation.
  • NCT01263041 -- MK-677 (oral GH secretagogue) in Alzheimer's disease, investigating GH/IGF-1 axis modulation in neurodegeneration.

Research outlook: While CJC-1295 DAC itself is unlikely to re-enter formal clinical development, it remains the benchmark long-acting GHRH analog in research. The success of tesamorelin has validated the GHRH analog therapeutic class, and next-generation long-acting GHRH analogs may build on the DAC bioconjugation concept.

Comparison to Related Compounds

CompoundHalf-lifeGH PatternMechanismFDA Status
CJC-1295 DAC~6-8 daysSustained elevation with preserved pulsatilityGHRH-R agonist + albumin conjugationNot approved
Mod GRF 1-29 (CJC-1295 no DAC)~30 minutesDiscrete pulsatileGHRH-R agonistNot approved
Sermorelin~10-12 minutesPulsatile (brief)GHRH-R agonist (native GRF 1-29)FDA-approved (discontinued)
Tesamorelin (Egrifta)~26-38 minutesPulsatileGHRH-R agonist (trans-3-hexenoic acid modified)FDA-approved (HIV lipodystrophy)
MK-677 (ibutamoren)~24 hoursSustained elevationGHS-R1a agonist (oral, non-peptide)Not approved
Ipamorelin~2 hoursPulsatileGHS-R1a agonist (selective GHRP)Not approved

Key distinctions:

  • CJC-1295 DAC vs Mod GRF 1-29: The DAC moiety extends half-life from ~30 minutes to ~6-8 days through covalent albumin binding. This enables weekly rather than multiple-daily dosing but produces sustained rather than discrete GH pulses. Mod GRF 1-29 more closely mimics physiological GHRH pulsatility, while CJC-1295 DAC provides continuous GH axis stimulation with greater convenience.
  • CJC-1295 DAC vs sermorelin: Sermorelin is unmodified GHRH (1-29) with a half-life under 12 minutes, requiring multiple daily injections for meaningful GH elevation. CJC-1295 DAC achieves equivalent or greater GH stimulation with weekly dosing. Sermorelin was FDA-approved but commercially discontinued due to supply issues, not safety concerns.
  • CJC-1295 DAC vs tesamorelin: Tesamorelin is the only GHRH analog currently FDA-approved (for HIV-associated lipodystrophy). It has a slightly longer half-life than sermorelin (~26-38 min) but still requires daily dosing. Tesamorelin has demonstrated reduction in visceral adipose tissue and hepatic fat (Stanley et al., 2015).
  • CJC-1295 DAC vs MK-677: MK-677 is an oral, non-peptide ghrelin receptor agonist with a ~24-hour half-life. It acts through the GHRP/ghrelin pathway rather than the GHRH pathway, making it mechanistically complementary to CJC-1295 DAC. MK-677 is more convenient (oral dosing) but may increase appetite and cortisol through ghrelin-mediated effects.

Safety Profile

In clinical studies, CJC-1295 DAC has been generally well tolerated. Reported side effects include injection site reactions (redness, swelling), transient flushing, and headache. Because it preserves physiologic pulsatile GH release rather than producing supraphysiologic continuous elevation, the side effect profile is considered more favorable than that of exogenous GH. However, long-term safety data in humans remains limited. As with all GH secretagogues, potential concerns include effects on glucose metabolism and theoretical risks related to sustained IGF-1 elevation.

Pharmacokinetic Profile

CJC-1295 with DAC — Pharmacokinetic Curve

Subcutaneous injection
0%25%50%75%100%0m7d14d21d28d35dTimeConcentration (% peak)T_max 33.6hT_1/2 7d
Half-life: 7dT_max: 24hDuration shown: 35d

Quick Start

Typical Dose
1-2mg weekly
Frequency
Once or twice weekly (e.g., Monday/Thursday for split dosing)
Route
Subcutaneous injection
Cycle Length
8-12 weeks
Storage
Lyophilized: 2-8°C refrigerated; Reconstituted: 2-8°C refrigerated, use within 30 days

Molecular Structure

2D Structure
CJC-1295 with DAC molecular structure
Molecular Properties
Formula
C152H252N44O42
Weight
3 Da
Length
30 amino acids
CAS
863288-34-0
PubChem CID
91976842
Exact Mass
3366.8969 Da
LogP
-10.7
TPSA
1450 Ų
H-Bond Donors
52
H-Bond Acceptors
48
Rotatable Bonds
118
Complexity
7710
Identifiers (SMILES, InChI)
InChI
InChI=1S/C152H252N44O42/c1-22-79(15)118(194-125(214)84(20)171-135(224)107(68-115(206)207)181-124(213)81(17)169-126(215)91(155)65-87-41-45-89(201)46-42-87)147(236)189-106(66-86-34-25-24-26-35-86)141(230)196-120(85(21)200)149(238)180-99(51-54-114(158)205)132(221)190-111(72-199)145(234)185-105(67-88-43-47-90(202)48-44-88)140(229)178-96(40-33-59-168-152(164)165)128(217)177-94(37-28-30-56-154)133(222)193-117(78(13)14)146(235)187-100(60-73(3)4)134(223)170-82(18)123(212)175-97(49-52-112(156)203)130(219)183-103(63-76(9)10)138(227)191-109(70-197)143(232)172-83(19)122(211)174-95(39-32-58-167-151(162)163)127(216)176-93(36-27-29-55-153)129(218)182-102(62-75(7)8)137(226)184-101(61-74(5)6)136(225)179-98(50-53-113(157)204)131(220)186-108(69-116(208)209)142(231)195-119(80(16)23-2)148(237)188-104(64-77(11)12)139(228)192-110(71-198)144(233)173-92(121(159)210)38-31-57-166-150(160)161/h24-26,34-35,41-48,73-85,91-111,117-120,197-202H,22-23,27-33,36-40,49-72,153-155H2,1-21H3,(H2,156,203)(H2,157,204)(H2,158,205)(H2,159,210)(H,169,215)(H,170,223)(H,171,224)(H,172,232)(H,173,233)(H,174,211)(H,175,212)(H,176,216)(H,177,217)(H,178,229)(H,179,225)(H,180,238)(H,181,213)(H,182,218)(H,183,219)(H,184,226)(H,185,234)(H,186,220)(H,187,235)(H,188,237)(H,189,236)(H,190,221)(H,191,227)(H,192,228)(H,193,222)(H,194,214)(H,195,231)(H,196,230)(H,206,207)(H,208,209)(H4,160,161,166)(H4,162,163,167)(H4,164,165,168)
InChIKeyXOZMWINMZMMOBR-UHFFFAOYSA-N

Research Indications

Growth Hormone

Strong Evidence
Sustained GH Elevation

Continuous growth hormone release for 6-8 days per injection.

Strong Evidence
IGF-1 Optimization

Significantly elevates IGF-1 levels for extended periods.

Good Evidence
Convenience Protocol

Weekly dosing ideal for simple administration.

Body Composition

Good Evidence
Fat Loss Enhancement

Continuous GH elevation promotes lipolysis and fat metabolism.

Good Evidence
Lean Mass Gains

Sustained anabolic environment supports muscle protein synthesis.

Anti-Aging

Moderate Evidence
Recovery Enhancement

Accelerated healing and recovery from exercise or injury.

Research Protocols

subcutaneous Injection

Long-acting GHRH analog with Drug Affinity Complex. Administered once weekly at bedtime on an empty stomach.

GoalDoseFrequency
Conservative1,000 mcg (1 mg)Once weekly
Standard2,000 mcg (2 mg)Once weekly
Reconstitution Guide (2mg vial + 1mL BAC water)
  1. Wipe vial tops with alcohol swab
  2. Draw 1.0 mL bacteriostatic water into syringe
  3. Inject slowly down the inside wall of the peptide vial
  4. Gently swirl to dissolve — never shake
  5. Resulting concentration: 2.0 mg/mL
  6. For 1,000 mcg (1 mg) dose: draw 50 units (0.50 mL)
  7. For 2,000 mcg (2 mg) dose: draw 100 units (1.00 mL)
  8. Store reconstituted vial refrigerated at 2-8°C

Interactions

Peptide Interactions

GHRELINcompatible
  • CJC-1295 DAC vs MK-677: MK-677 is an oral, non-peptide ghrelin receptor agonist with a ~24-hour half-life.

What to Expect

What to Expect

Week 1

Possible water retention, improved sleep, increased appetite

Week 2-3

Noticeable recovery improvements, some report joint discomfort

Week 4-6

Visible body composition changes, increased vascularity

Week 6-8

Continued improvements but watch for desensitization signs

Post-cycle

Most gains maintained; IGF-1 returns to baseline in 2-3 weeks

Safety Profile

Common Side Effects

  • Water retention
  • Joint pain
  • Carpal tunnel symptoms

Contraindications

  • Diabetes history
  • Cancer history
  • Predisposed sleep apnea

Discontinue If

  • Severe joint pain or carpal tunnel syndrome
  • Excessive water retention affecting daily life
  • Numbness or tingling in extremities
  • Significant blood glucose dysregulation
  • Signs of acromegaly (jaw growth, hand/feet enlargement)
  • Persistent lethargy indicating adrenal effects

Quality Indicators

What to look for

  • High purity requirement (>98%); impurities cause more side effects
  • Proper DAC labeling; legitimate products clearly state 'with DAC'

Caution

  • Higher cost than non-DAC (2-3x more due to complex manufacturing)
  • Foaming during mixing is normal for DAC peptides; wait for foam to settle

Red flags

  • Extremely cheap pricing (if priced similar to non-DAC, likely mislabeled)
  • Clear solution immediately (should be slightly cloudy initially)

Frequently Asked Questions

References (8)

  1. [1]
    Prolonged Growth Hormone Elevation with CJC-1295 DAC (2006)
  2. [2]
    Dose-Response Relationship of CJC-1295 DAC (2008)
  3. [3]
    Long-term Effects on GH/IGF-1 Axis (2012)
  4. [4]
    Comparative Pharmacokinetics: DAC vs Non-DAC Variants (2015)
  5. [5]
  6. [6]
  7. [7]
  8. [8]
Updated 2026-03-08Sources: jabronistore-wiki, peptide-wiki-mdx, pep-pedia, pubchem, peptide-wiki-mdx-v2

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